RU58841 has not been approved anywhere in the world for hair loss treatment. Some people believe it (or RU-58841, or RU) is a safe alternative to finasteride. But is it really true? Are there any better options available if someone does not want to use finasteride? What is the verdict on how often RU should be used? What are the caveats when one tries to concoct their own solution? This blog will try to answer all of these and many other commonly asked questions about this treatment from a
Compounding pharmacy may offer a concortion solution that has a combination of any of the following ingredients: minoxidil with concentration 2% to 15%, finasteride typically at 0.1% or 0.2%, azelaic acid varing from 1% to 12.5%, ketoconazole in the range of 1% to 2%, progesterone typically at 1% to 2.5%.
Because the solution is cusomized and it is labor intensive and requires special skill to compound, it comes with a great price tag, it generally costs over 100 bucks per 60 mL solution.
Finasteride pill (brand name Propecia) is one of the only two FDA approved products for hair loss treatment. Topical finasteride has been explored by many users to lower side effects of finasteride pill. This blog will discuss (1) if the topical finasteride works, (2) whether it has fewer side effects, (3) the best way to avoid side effects, (4) how to compound topical finasteride yourself, and (5), how to maximize the effects of topical finasteride. Other commonly asked questions will also be discussed.
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Ketoconazole is approved for use as an anti-fungal agent in the treatment of infections that occur due to fungi and yeasts. This review is focus on the its usage in shampoo for hair loss treatment.
How Does It Work? As far as its role in treating and preventing hair loss is concerned, there are a number of theories. According to the American Hair Loss Association, apart from its anti-fungal properties it also has anti-androgenic effects. When ingested orally it is known to inhibit the binding of androgens to receptors in the body, which would include the binding of DHT to hair-follicle receptors (see the ‘Hair Loss Explained’ section for the leading causal theory of hair loss). It may