Some people believe RU58841 (or RU-58841, or RU) is a safe alternative to finasteride. This blog will examine if it is true. A procedure for making your own solution is also provided.
RU58841, also known as PSK-3841 or HMR-3841, is a nonsteroidal anti-androgen (NSAA) which was investigated and developed by ProStrakan (a Scottish based speciality pharmaceutical company) for its potential therapeutic applications in conditions which were driven by androgens, such as acne, hirsutism (excessive hair growth) and
Trinov Lozione Anticaduta, a hair loss product launched in 2019 by an Italian company Fidia Farmaceutic
Compounding pharmacy may offer a concortion solution that has a combination of any of the following ingredients: minoxidil with concentration 2% to 15%, finasteride typically at 0.1% or 0.2%, azelaic acid varing from 1% to 12.5%, ketoconazole in the range of 1% to 2%, progesterone typically at 1% to 2.5%.
Because the solution is cusomized and it is labor intensive and requires special skill to compound, it comes with a great price tag, it generally costs over 100 bucks per 60 mL solution.
Finasteride pill (brand name Propecia) is one of the only two FDA approved products for hair loss treatment, the other being minoxidil. Topical finasteride has been explored by many users to lower side effects of finasteride pill. This blog will discuss (1) if the topical finasteride works, (2) whether its side effects is less than oral finasteride, (3) what is the best way to avoid side effects, and (4) how to compound topical finasteride yourself.
Ketoconazole is approved for use as an anti-fungal agent in the treatment of infections that occur due to fungi and yeasts. This review is focus on the its usage in shampoo for hair loss treatment.
How Does It Work? As far as its role in treating and preventing hair loss is concerned, there are a number of theories. According to the American Hair Loss Association, apart from its anti-fungal properties it also has anti-androgenic effects. When ingested orally it is known to inhibit the binding of androgens to receptors in the body, which would include the binding of DHT to hair-follicle receptors (see the ‘Hair Loss Explained’ section for the leading causal theory of hair loss). It may